Future work

a. Using GFP or other reporter genes to better understand the subcellular localization of Gcn5 in the absence of Ada2 and Ada3.

b. To investigate whether defect of HAT module affects the function of other modules in SAGA complex.

c. Using protein-protein interaction to elucidate whether other proteins function as Gcn5 will interact with SAGA complex in the absence of Gcn5.

d. To find which transcription factor is regulated by SAGA complex against oxidative stress in C. glabrata.

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