• 沒有找到結果。

民國九十年台灣地區癌症主要死亡原因

N/A
N/A
Protected

Academic year: 2021

Share " 民國九十年台灣地區癌症主要死亡原因"

Copied!
5
0
0

加載中.... (立即查看全文)

全文

(1)

參 考 文 獻

民國九十年台灣地區主要死亡原因。行政院衛生署。

民國九十年台灣地區癌症主要死亡原因。行政院衛生署。

賴羿如、項千芸、侯庭鏞。肝細胞轉形之分子機制及分子療程之探討。中國醫藥學院醫學研究所 碩士論文(2000)。

Adlercreutz, H, 1990. Western diet and Western diseases: some hormonal and biochemical mechanisms and associations. Scand. Clin. Lab. Invest. Suppl. 201, 3-23.

Angel, P., Imagawa, M., Chiu, R., Stein, B., Imbra, R.J., Rahmsdorf, H.J., Jonat, C., Herrlich, P., and Karin, M. 1987. Phorbol ester-inducible genes contain a common cis element recognized by a TPA-modulated trans-acting factor. Cell 49: 729-739.

Angel, P., and Karin, M. 1991. The role of Jun, Fos, and the AP-1 complex in cell proliferation and transformation. Biochim. Biophy. Acta. 1072: 129-157.

Befeler, A.S. and Bisceglie, A.M.D., 2002. Hepatocellular carcinoma: diagnosis and treatment.

Gastroenterology 122:1609-1619

Bernstein, L.R., and Colburn, N.H. 1989. AP1/jun function is differentially induced in promotion-sensitive and resistant JB6 cells. Science 244: 566-569.

Birt, DF, Mitchell, D, Gold, B, Pour, P, Pinch, HC, et al.1997. Inhibition of ultraviolet light-induced skin carcinogenesis in SKH-1 mice by apigenin, a plant flavonoid. Anticancer Res. 17, 85-91.

Bismuth, H., Chiche, L., Castaing, D. 1995. Surgical treatment of hepatocellular carcinomas in noncirrhotic liver: experience with 68 liver resections. World J. Surg.19:35–41.

Bruce A . et al., 2002, Molecular biology of the cell, 4th edition. pp 983-1026.

Buja, L.M., Ferrans, V.J., Mayers, R.J., Robert, W.C., Henderson, E.S., 1973. Cardiac ultrastructural changes induced by daunorubicin therapy. Cancer 32, 771-/778.

Caitagirone, S, Rossi, C, Poggi, A., Ranelletti, F.O., Natali, P.G. et al. 2000. Flavonoids apigenin and quercitin inhibit melanoma growth and metastatic potential. Int. J. Cancer. 87, 595-600.

Carroll, K.K., Guthrie, N., So, F.V., and Chambers, A.F. 1998. Anticancer properties of flavonoids, with emphasis on citrus flavonoids. Flavonoids in health and disease. pp 437-446.

Chaumontet, C., Bex, V., Gaillard-Sanchez, I., Seillan-Heberden, C., Suschete, M. et al. 1994.

Apigenin and tangeretin enhance gap junction intercellular communication in rat liver epithelial cells.

Carcinogenesis 15, 2325-2330.

Cody, V. , Middleton, E., Harborne, J.B., and Beretz, A. 1988. Plant flavonoids in biology and Medicine II: Biochemical, Cellular and Medicinal Properties. Alan R. Liss Inc., New York.

Diplock, A.T., Charleux, J.L., Crfozier-Willi, G., Kok, F.J., Rice-Evans, C., Roberfroid, M., Stahl, W., Vina-Ribes, J., 1998. Functional food science and defence against reactive oxygen species. Br. J.

Nutr. 80, 77-/112.

Domann, F.E., Levy, J.P., Birrer, M.J., and Bowden, G.T. 1994. Stable expression of a c-JUN deletion mutant in two malignant mouse epidermal cell lines blocks tumor formation in nude mice. Cell

(2)

Growth Differ. 5: 9-16.

Dong, Z., Birrer, M.J., Watts, R.G., Matrisian, L.M., and Colburn, N.H. 1994. Blocking of tumor promoter-induced AP-1 activity inhibits induced transformation in JB6 mouse epidermal cells. Proc.

Natl. Acad. Sci. 91: 609-613.

Elledge S.J., and Harper J.W. 1994. Cdk inhibitors: on the threshold of checkpoints and development.

Curr. Opin. Cell Biol. 6: 847-852.

Fornari, F.A., Randolph, J.K., Yalowich, J.C., Ritke, M.K., Gewirtz, D.A. 1994. Interference by doxorubicin with DNA unwinding in MCF-7 breast tumour cells. Mol. Pharmacol. 45, 649-/656.

Gabor, M. 1979. Anti-inflammatory substances of plant origin, in handbook of experimental pharmacology: Anti-inflammatory drugs. pp698-739. Spinger-Verlag, New York.

Gabor, M. 1986. The pharmacology of benzopyrone derivatives and related compounds, Akademiai Kiad, Budapest.

Gewirtz, D.A. 1999. A Critical evaluation of the mechanisms of action proposed for the antitumor effects of the anthracycline antibiotics adriamycin and daunorubicin. Biochem. Pharmacol. 57, 727-/741.

Goldberg Y, Nassif II, Pittas A, Tsai L.L., Dynlacht B.D., Rigas B., and Shiff S.J. 1996. the anti-prolferative effect of sulindac and sulindac sulfide on HT-29 colon cancer cells: alternations in tumor suppressor and cell cycle-regulatory proteins. Oncogene 12: 893-901.

Graves, P.R., Yu, L., Schwarz, J.K., Gales, J., Sausville, E.A., O’Connor, P.M., and Piwnica-Worms, H.

2000. The chk1 protein kinase and the cdc25C regulatory pathways are targets of the anticancer agent UCN-01. J. Biol. Chem., 275: 5600-5605.

Gupta, S., Barrett, T., Whitmarsh, A.J., Cavanagh, J., Sluss, H.K., Derijard, B., and Davis, R.J. 1996.

Selective interaction of JNK protein kinase isoforms with transcription factors. EMBO J. 15:

2760-2770.

Gupta, S., Campbell, D., Dérijard, B., and Davis, R.J. 1995. Transcription factor ATF2 regulation by the JNK signal transduction pathway. Science 267: 389-393.

Halazonetis, T.D., Georgopoulos, K., Greenberg, M.E. and Leder, P. 1988. C-jun dimerizes with itself and c-fos forming complexes of different DNA binding abilities. Cell 55: 917-924.

Harborne, J.B. 1988a. The flavonoids: Advances in Research Since 1986. Chapman & Hall, New York Harborne, J.B. 1988b. Flavonoids in the environment: Structure-activity relationships, in Plant

flavonoids in Biology and Medicine II: Biochemical Cellular and Medicinal Properties. pp 17-28, Alan R. Liss, Inc: New York.

Harbury, P.B., Zhang, T., Kim, P.S., and Alber, T. 1993. A switch between two-, three-, and four-stranded coiled coils in GCN4 leucine zipper mutants. Science 262: 1401-1407.

Harrison’s Principles of Internal Medicine, 14th Ed. pp 579-580

Hartwell, L. H., and Kastan, M. B. 1994. Cell cycle control and cancer. Science, 266:1821-1828.

Havsteen, B. 1984. Flavonoids: A class of natural products of high pharmacological potency. Biochem.

Pharmacol. 32: 1141-1148.

Herschman, H.R. 1991. Primary response genes induced by growth factors and tumor promoters. Ann.

Rev. Biochem. 60: 281-319.

(3)

Ichinose, Y., Takanashi, N., Yano, T., 1995. A phase III trial of oral tegafur and uracil p lus cisplatin in patients with inoperable nonsmall cell lung cancer. Cancer 75, 2677-2680.

Jia, S.S., Ma, C.M., Li, Y.H., Hao, J.H. 1992. Glycosides of phenolic acid and flavonoids from the leaves of Glycyrrhiza uralensis Ficsh. Yao Xue Xue Bao. 27(6):441-4.

Jochum, W., Passegue, E., Wagner, E.F. 2001. AP-1 in mouse development and tumorigenesis.

Oncogene 30;20(19):2401-12.

Kaufmann, W.K., Levedakuo, E.N., Grady, H.L., Paules, R.S., and Stein, G.H. 1995. attenuation of G2 checkpoint function precedes human cell immortalization. Cancer Res. 55: 7-11,.

Koo, H., Rosalen, P.L., Cury, J.A., Park, Y.K., Bowen, W.H. 2002. Effects of compounds found in propolis on Streptococcus mutans growth and on glucosyltransferase activity. Antimicrob. Agents Chemother. 46(5):1302-9.

Kuo, M.L., Lee, K.C., and Lin, J.K. 1992. Genotoxicities of nitropyrenes and their modulation by apigenin, tannic acid, ellagic acid and indole-3-carbinol in the Salmonella and CHO systems. Mutat.

Res. 270, 87-95.

Lamb, R.F., Hennigan, R.F., Turnbull, K., Katsanakis, K.D., MacKenzie, E.D., Birnie, G.D., and Ozanne, B.W. 1997. AP-1-mediated invasion requires increased expression of the hyaluronan receptor CD44. Mol. Cell Biol. 17: 963-976.

Landschulz, W.H., Johnson, P.F., and McKnight, S.L. 1988. The leucine zipper: a hypothetical structure common to a new class of DNA binding proteins. Science 240: 1759-1764.

Lefrak, E.A., Pitha, J., Rosenheim, S., Gottleib, J.A., 1973. A clinicopathologic analysis of adriamycin cardiotoxicity. Cancer 32, 302-/314.

Lepley, D.M., Li, B., Birt, D.F. and Pelling, J.C.. 1996. The chemopreventive flavonoid apigenin induces G2/M arrest in keratinocytes. Carcinogenesis 17: 2367-2375,.

Li, J.J., Rhim, J.S., Schlegel, R., Vousden, K.H., and Colburn, N.H. 1998. Expression of dominant negative Jun inhibits elevated AP-1 and NF -kappa B transactivation and suppresses anchorage independent growth of HPV immortalized human keratinocytes. Oncogene 16: 2711-2721.

Mac Vean, M., Weinberg, W.C., and Pelling, J.C. 2002. Ap21waf1-independent pathway for inhibitory phosphorylation of cyclin-dependent kinase p34cdc2 and concomitant G2-M arrest by the chemopreventive flavonoid apigenin. Mol. Carcinogenesis 33: 36-43,.

Mac Vean, M, Xiao, H, Isobe, K, and Pelling, J.C. 2000. increase in wild-type p53 stability and transactivational activity by the chemopreventive agent apigenin in keratinocytes. Carcinogenesis 21, 633-639.

Middleton, E. 1984. The flavonoids. Trends. Pharmacol. Sci. 277, 375-381.

Middleton, E., and Kandaswami, C. 1993. The impact of plant flavonoids on mammalian biology:

Implications for immunity, inflammation and cancer, in The Flavonoids: Advances in Research Science 1986 (Harborne IR ed) pp 619-645. Chapman and Hall, London.

Moragoda, L., Jaszewski, R., and Majumdar, A.P. 2001. Curcumin induced modulation of cell cycle and apoptosis in gastric and colon cancer cells. Anticancer Res. 21:873-878.

O'Neil, K.T., Hoess, R.H., and DeGrado, W.F. 1990. Design of DNA-binding peptides based on the leucine zipper motif. Science 249: 774-778.

(4)

Sadzuka, Y., Mochizuki, E., Takino, Y., 1995. Mechanism of caffeine modulation of the antitumor activity of adriamycin. Toxicol. Lett. 75, 35-49.

Sato, F., Matsukawa, Y., Matsumoto, K., Nishino, H., and Sakai, T. 1994. Apigenin induces morphological differentiation and G2-M arrest in rat neural cells. Biochem. and Biophys. Res. Com.

204: 578-584.

Shapiro, G.I., Koestner, D.A., Matranga, C.B., and Rollins, B.J. 1999. Flavopiridol induces cell cycle arrest and P53-independent apoptosis in non-small cell lung cancer cell lines. Clin. Cancer Res. 5:

2925-2938,.

Sheer C.J. 1996. cancer cell cycle. Science 274: 1672-1677.

Shuman, J.D., Vinson, C.R., and McKnight, S.L. 1990. Evidence of changes in protease sensitivity and subunit exchange rate on DNA binding by C/EBP. Science 249: 771-774.

Singal, P.K., Deally, C.M.R., Weinberg, L.E., 1987. Subcellular effects of adriamycin in the heart: a concise review. J. Mol. Cell. Cardiol. 19, 817-/828.

Singal, P.K., Iliskovic, N., 1998. Adriamycin cardiomyopathy. N. Engl. J. Med. 339, 900-/905.

Singal, P.K., Li, T., Kumar, D., Danelisen, I., Iliskovic, N., 2000. Adriamycin-induced heart-failure:

mechanism and modulation. Mol. Cell. Biochem. 207, 77-/85.

Southern, P.J., and Berg, P. 1982. Transformation of mammalian cells to antibiotic resistance with a bacterial gene under control of the SV40 early region promoter. J. Mol. Appl. Genet. 1: 327-341.

Stochmal, A, Piacente, S, Pizza, C, De Riccardis , F, Leitz, R, Oleszek, W. 2001. Alfalfa (Medicago sativa L.) flavonoids. 1. Apigenin and luteolin glycosides from aerial parts. J. Agric. Food Chem.

49(2):753-8.

Taylor, I.C., and Kingston, R.E. 1990. Factor substitution in a human HSP70 gene promoter:

TATA-dependent and TATA -independent interactions. Mol. Cell Bio. 10: 165-175.

Taylor, W.R., and Stark, G.R. 2001. Regulation of the G2-M transition by p53. Oncogene, 20:

1803-1815.

Tyagi, A.K., Singh, R.P., Agarwal, C., Chan, D.C.F., and Agarwal, R. 2002. Silibinin strongly synergizes human prostate carcinoma DU145 cells to doxorubicin-induced growth inhibition, G2-M arrest, and apoptosis. Clin. Cancer Res. 8:3512-3519.

Van Acker, S.A.B.E., Kramer, K., Grimbergen, J.A., Van der Berg, D.J., Van der Vijgh, W.J.F., Bast, A., 1995. Monohydroxyethylrutoside a protector against chronic doxorubicin-induced cardiotoxicity.

Br. J. Pharmacol. 115, 1260-1264.

Van Acker, S.A.B.E., Boven, E., Juiper, K., Van der Berg, D.J., Grimbergen, J.A., Kramer, K., Bast, A., Van der Vijgh, W.J.F., 1997. Monohydroxyethylrutoside is a dose dependent cardioprotective agent and does not affect the antitumor activity of doxorubicin. Clin. Cancer Res. 3, 1747-1754.

Vinson, C.R., Sigler, P.B., and McKnight, S.L. 1989. Scissors -grip model for DNA recognition by a family of leucine zipper proteins. Science 246: 911-916.

Von Hoff, D.D., Layard, M.W., Basa, P., Davis, H.L., Von Hoff, A.L., Rozwncweig, M., and Muggia, F.

M.: Risk factors for doxorubicin -induced congestive heart failure. Ann. Intern. Med. 91: 710-717, 1979.

Wang, C, and Kurzer, M.S. 1997. Phytoestrogen concentration determines effects on DNA synthesis in

(5)

human breast cancer cells. Nutr. Cancer 28, 236-247.

Wang, I.K., Lin-Shiau, S.Y., and Lin, J.K. 1999. induction of apoptosis by apigenin and related flavonoids through cytochrome C release and activation of caspas e-9 and caspase-3 in leukemia HL-60 cells. Eur. J. Cancer 35, 1517-1525.

Wang, W, Heideman, L, Chung, C.S., Pelling, J.C., Koehler, K.J. et al. 2000. Cell cycle arrest at G2-M and growth inhibition by apigenin in human colon carcinoma cell lines. Mol. Carcinog. 28, 102-110.

Wang, W., Heideman, L., Chung, C.S., Pelling, J.C., Koehler, K.J. and Birt, D.F. 2000. Cell-cycle arrest at G2-M and growth inhibition by apigenin in human colon carcinoma cell line. Mol. Carcinog.

28:102-110.

Yin, F., Giuliano, A.E., Law, R.E., and Van Herle, A.J. 2001. Apigenin inhibits growth and induces G2/M arrest by modulating cyclin-CDK regulators and ERK MAP kinase activation in breast carcinoma cells. Anticancer Res. 21: 413-420.

參考文獻

相關文件

Incubation with bisphosphonates reduced the cell count, but only became significant for zoledronate at 50 µmol (p=0.028) compared to the control; incubation with 50 µmol

原文題目(出處): Bisphosphonates: restrictions for vasculogenesis and angiogenesis: inhibition of cell function of endothelial progenitor cells and mature endothelial cells

The clinical and radiological features of 6 cases of NBCCS were characterized into major and minor criteria and compared with features reported in Indian patients and in patients

Human papillomavirus- related squamous cell carcinoma of the oropharynx: a comparative study in whites and African Americans. Prevalence of human papillomavirus type 16 DNA in

We describe the clinical, macroscopic, cytological, histopathological, immunohistochemical, serodiag- nostic and aspects of pemphigus vulgaris (PV) in the oral gingiva

Central granular cell odontogenic tumour, report of the first malignant case and review of the literature. Human leucocyte antigen typing in

6 M 30 Palmar and/or plantar pits, skin cysts, one basal cell carcinoma, milia, neurofibroma, jaw cysts, calcification of the falx cerebri, scoliosis, missing/malformed

Samples of oral fibrous hyperplasia were used as positive controls for E-cadherin and vimentin staining. In addition, samples of normal cardiac striated muscle and